Department of Biochemical Engineering, Hohhot Vocational College, Inner Mongolia 010051, China
Introduction: The identification and characterization of the complex regulatory mechanism system for determining the cell fate is particularly critical to the identification and characterization of the mitochondrial DNA genetic polymorphisms of cross-type cells.
Materials and methods: The aim of this study was to explain the process of MtDNA DNA polymorphism in marine animal and its mechanism.
Result: Each time the germ cells are formed, the polymorphism of mitochondrial DNA in the South China Sea Marine animals is edited, that is, the original mitochondrial DNA polymorphism pattern is reconstructed. High levels of plasma homocysteine inhibit DNA methyltransferase, resulting in low mitochondrial DNA genetic polymorphism. Low polymorphism of DNA mitochondrial DNA alters smooth muscle cell proliferation, causes endothelial cell dysfunction, and increases the expression of inflammatory mediators. Mitochondrial DNA of Marine animals in the South China Sea has the characteristics of DNA genetic diversity in the evaluation of health, and the polymorphism of mitochondrial DNA of Marine animals only changes the epigenetic inheritance. Cell type-specific low mitochondrial DNA polymorphism markers are associated with cell type-specific super-enhancers that drive cell identity gene expression. The framework provides a powerful explanation for the key functions and features of different types of cell-specific low-mitochondrial DNA genetic polymorphisms.
Conclusion: Finding the corresponding marine animal mtDNA Polymorphism elements will be of great help to the diagnosis, treatment, and prognosis of all kinds of diseases.
Health evaluation, Marine Animal, Mitochondria, DNA, Genetic polymorphism.